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Physiological stress exacerbates murine colitis by enhancing proinflammatory cytokine expression that is dependent on IL-18
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Furthermore, treatment of rat hippocampal cells and SHSY5Y human neuroblastoma cells with H 2 O 2 at the early stages of oxidative stress exposure results in tau dephosphorylation at the tau1 epitope by CDK5 via Pin1 activation (203)
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