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We have found that Sema4D-induced nuclear translocation of AR is dependent on Ran signalling and the GAP domain of PlexinB1, suggesting that PlexinB1/B2 contribute to ADT resistance through their function as RanGAPs

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Here, we discuss whether the loss of nutrients associated with disc degeneration limits the effectiveness of biologic approaches, and indicate that this neglected problem requires investigation if clinical application of such therapies is to succeed