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These mechanisms also modulate neuroimmune responses by reducing the release of pro-inflammatory cytokines (such as TNF-α and IL-1β) from immune cells, and by influencing the activity of astrocytes and microglia, both of which are implicated in MS-related neuroinflammation [15,16,24,25]
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Symptoms such as these may be associated with an increased risk for suicidal thinking and behavior and indicate a need for very close monitoring and possibly changes in the medication
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