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Because an adequate amount of H 2 O 2 is required to initiate GSIS [131], the Sod1-deficient islets might not produce sufficient H 2 O 2 or have appropriate ratios of H 2 O 2 to superoxide to support GSIS
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Despite advancements in molecular typing and targeted therapies that have improved the prognosis of some subtypes, the mechanisms driving AML heterogeneity and its microenvironmental interactions remain poorly understood (215)
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